Search results for "Protein–DNA interaction"

showing 5 items of 5 documents

Oxidatively generated base modifications in DNA: Not only carcinogenic risk factor but also regulatory mark?

2016

The generation of DNA modifications in cells is in most cases accidental and associated with detrimental consequences such as increased mutation rates and an elevated risk of malignant transformation. Accordingly, repair enzymes involved in the removal of the modifications have primarily a protective function. Among the well-established exceptions of this rule are 5-methylcytosine and uracil, which are generated in DNA enzymatically under controlled conditions and fulfill important regulatory functions in DNA as epigenetic marks and in antibody diversification, respectively. More recently, considerable evidence has been obtained that also 8-oxo-7,8-dihydroguanine (8-oxoG), a frequent pro-mu…

0301 basic medicineGuanineDNA RepairTranscription GeneticDNA repairCarcinogenesisBiochemistryDNA GlycosylasesEpigenesis Genetic03 medical and health sciencesRisk FactorsPhysiology (medical)NeoplasmsAnimalsGuanine Nucleotide Exchange FactorsHumansProtein–DNA interactionTranscription factor030102 biochemistry & molecular biologybiologyBase excision repairDNAProliferating cell nuclear antigenOxidative Stress030104 developmental biologyHistoneBiochemistryDNA glycosylasebiology.proteinOxidation-ReductionNucleotide excision repairSignal TransductionFree radical biologymedicine
researchProduct

Excision of Uracil from Transcribed DNA Negatively Affects Gene Expression

2014

Uracil is an unavoidable aberrant base in DNA, the repair of which takes place by a highly efficient base excision repair mechanism. The removal of uracil from the genome requires a succession of intermediate products, including an abasic site and a single strand break, before the original DNA structure can be reconstituted. These repair intermediates are harmful for DNA replication and also interfere with transcription under cell-free conditions. However, their relevance for cellular transcription has not been proved. Here we investigated the influence of uracil incorporated into a reporter vector on gene expression in human cells. The expression constructs contained a single uracil opposi…

DNA RepairTranscription GeneticGreen Fluorescent ProteinsGene ExpressionDNA and ChromosomesBiologyBiochemistryCell LineDNA Glycosylaseschemistry.chemical_compoundGenes ReporterActivation-induced (cytidine) deaminaseHumansheterocyclic compoundsProtein–DNA interactionAP siteUracilUracil-DNA GlycosidaseMolecular BiologyUracilDNACell BiologyBase excision repairMolecular biologyThymine DNA GlycosylasechemistryDNA glycosylaseGene Knockdown TechniquesUracil-DNA glycosylasebiology.proteinHeLa CellsNucleotide excision repairJournal of Biological Chemistry
researchProduct

Interactions between DNA damage, repair, and transcription

2010

This review addresses a variety of mechanisms by which DNA repair interacts with transcription and vice versa. Blocking of transcriptional elongation is the best studied of these mechanisms. Transcription recovery after damage therefore has often been used as a surrogate marker of DNA repair in cells. However, it has become evident that relationships between DNA damage, repair, and transcription are more complex due to various indirect effects of DNA damage on gene transcription. These include inhibition of transcription by DNA repair intermediates as well as regulation of transcription and of the epigenetic status of the genes by DNA repair-related mechanisms. In addition, since transcript…

GeneticsGenome instabilityDNA RepairTranscription GeneticbiologyDNA repairDNA damageHealth Toxicology and MutagenesisGenomic InstabilityProliferating cell nuclear antigenCell biologyHigh-mobility groupGene Expression RegulationTranscription (biology)Geneticsbiology.proteinHumansProtein–DNA interactionDNA mismatch repairMolecular BiologyDNA DamageSignal TransductionMutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
researchProduct

The elemental role of iron in DNA synthesis and repair

2017

Iron is an essential redox element that functions as a cofactor in many metabolic pathways. Critical enzymes in DNA metabolism, including multiple DNA repair enzymes (helicases, nucleases, glycosylases, demethylases) and ribonucleotide reductase, use iron as an indispensable cofactor to function. Recent striking results have revealed that the catalytic subunit of DNA polymerases also contains conserved cysteine-rich motifs that bind iron–sulfur (Fe/S) clusters that are essential for the formation of stable and active complexes. In line with this, mitochondrial and cytoplasmic defects in Fe/S cluster biogenesis and insertion into the nuclear iron-requiring enzymes involved in DNA synthesis a…

Iron-Sulfur Proteins0301 basic medicineDNA RepairDNA polymeraseDNA damageDNA repairIronBiophysicsDNA repairEukaryotic DNA replicationSaccharomyces cerevisiaeBiochemistryDNA GlycosylasesBiomaterials03 medical and health sciencesRibonucleotide ReductasesHumansProtein–DNA interactionRibonucleotide reductaseReplication protein Achemistry.chemical_classificationDNA ligaseDeoxyribonucleasesDNA synthesis030102 biochemistry & molecular biologybiologyIron deficiencyDNA HelicasesMetals and AlloysHelicaseDNAYeast030104 developmental biologyIron cofactorBiochemistrychemistryChemistry (miscellaneous)biology.proteinIron-sulfur clusterMetallomics
researchProduct

Sequence-specific and DNA structure-dependent interactions of Escherichia coli MutS and human p53 with DNA

2013

Many proteins involved in DNA repair systems interact with DNA that has structure altered from the typical B-form helix. Using magnetic beads to immobilize DNAs containing various types of structures, we evaluated the in vitro binding activities of two well-characterized DNA repair proteins, Escherichia coli MutS and human p53. E. coli MutS bound to double-stranded DNAs, with higher affinity for a G/T mismatch compared to a G/A mismatch and highest affinity for larger non-B-DNA structures. E. coli MutS bound best to DNA between pH 6 and 9. Experiments discriminated between modes of p53-DNA binding, and increasing ionic strength reduced p53 binding to nonspecific double-stranded DNA, but had…

chemistry.chemical_classificationDNA ligaseDNA clampHMG-boxBase pairEscherichia coli ProteinsOsmolar ConcentrationBiophysicsDNACell BiologyBiologyBiochemistryMutS DNA Mismatch-Binding ProteinDNA binding siteBiochemistrychemistryMutS-1Escherichia coliHumansNucleic Acid ConformationProtein–DNA interactionAmino Acid SequenceTumor Suppressor Protein p53Molecular BiologyReplication protein AAnalytical Biochemistry
researchProduct